The influence of extremely low frequency magnetic fields on cytoprotection and repair
Identifieur interne : 000C60 ( Istex/Checkpoint ); précédent : 000C59; suivant : 000C61The influence of extremely low frequency magnetic fields on cytoprotection and repair
Auteurs : John A. Robertson [Canada] ; Alex W. Thomas [Canada] ; Yves Bureau [Canada] ; Frank S. Prato [Canada]Source :
- Bioelectromagnetics [ 0197-8462 ] ; 2007-01.
English descriptors
- Teeft :
- Astroglial cells, Biochem, Biochem biophys, Bioelectrochemistry dicarlo, Bioelectromagnetics, Blockage, Blood flow, Blood vessels, Calcium concentration, Calcium imbalances, Canadian institutes, Cardiac, Cardiac cells, Cardiac protection, Cardiovascular, Cardiovascular disease, Cell biochem, Cell cultures, Cell death, Cell differentiation, Cellular stress, Cepaea nemoralis, Cerebral ischemia, Chick, Chick embryos, Collateral, Collateral blood, Committee guidelines, Conventional treatments, Cytoprotection, Daily exposures, Development challenge fund, Dicarlo, Drosophila melanogaster, Eld, Electromagnetic, Embryo, Energy stores, Free oxygen, Free oxygen radicals, Free radicals, Frequency electromagnetic, Frequency range, Gene activity, Gene expression, Gene expression levels, Goodman, Grant sponsor, Guideline, Gutzeit, Health care, Health research, Heat shock, Heat shock protein, Heat shock proteins, Heat shock response, Heat stress, Hela cells, High sars, Hsps, Human cells, Human leukocytes, Hypoxia, Increase none, Inducible form, Infarct size, Ischemia, Ischemiareperfusion, Ischemiareperfusion injuries, Ischemic, Ischemic conditions, Ischemic injuries, Ischemic injury, Ischemic insult, Ischemic penumbra, Ischemic strokes, Ischemic tissue, Katp channels, Land snail, Lawson health research institute, Leukocyte, Litovitz, Lower sars, Many studies, Maximal expression, Metabolic byproducts, Metal shielding, Microwave exposure, Mild stress, Mobile phone radiation, Mrna, Mussel immunocyte, Myocardial cells, Nascent proteins, Nematode, Nematode caenorhabditis elegans, Neurosci lett shupak, Nitric, Nitric oxide, Ontario research, Opioid, Opioid receptors, Opioids, Other proteins, Other systems, Oxide, Pain thresholds, Physiol, Prato, Preconditioning, Preconditioning studies, Promoter, Protective effect, Protective effects, Protective proteins, Protein synthesis, Receptor, Repair damage, Reperfusion, Reperfusion injury, Reperfusion insult, Reperfusion stage, Rest period, Right vessel, Robertson, Sars, Short term, Shupak, Small heat shock proteins, Small hsps, Stress proteins, Stress response, Stroke foundation, Superoxide, Superoxide radicals, Thrombolytic agents, Transgenic, Transgenic studies, Wound healing.
Abstract
Ischemia‐reperfusion injuries, such as those suffered from various types of cardiovascular disease, are major causes of death and disability. For relatively short periods of ischemia, much of the damage is potentially reversible and in fact, does not occur until the influx of oxygen during the reperfusion stage. Because of this, there is a window of opportunity to protect the ischemic tissue. Here, we review several mechanisms of protection, such as heat shock proteins, opioids, collateral blood flow, and nitric oxide induction, and the evidence indicating that magnetic fields may be used as a means of providing protection via each of these mechanisms. While there are few studies demonstrating direct protection with magnetic field therapies, there are a number of published reports indicating that electromagnetic fields may be able to influence some of the biochemical systems with protective applications. Bioelectromagnetics © 2006 Wiley‐Liss, Inc.
Url:
DOI: 10.1002/bem.20258
Affiliations:
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unit="page-count">15</biblScope><publisher>Wiley Subscription Services, Inc., A Wiley Company</publisher><pubPlace>Hoboken</pubPlace><date type="published" when="2007-01">2007-01</date></imprint><idno type="ISSN">0197-8462</idno></series></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0197-8462</idno></seriesStmt></fileDesc><profileDesc><textClass><keywords scheme="Teeft" xml:lang="en"><term>Astroglial cells</term><term>Biochem</term><term>Biochem biophys</term><term>Bioelectrochemistry dicarlo</term><term>Bioelectromagnetics</term><term>Blockage</term><term>Blood flow</term><term>Blood vessels</term><term>Calcium concentration</term><term>Calcium imbalances</term><term>Canadian institutes</term><term>Cardiac</term><term>Cardiac cells</term><term>Cardiac protection</term><term>Cardiovascular</term><term>Cardiovascular disease</term><term>Cell biochem</term><term>Cell cultures</term><term>Cell death</term><term>Cell differentiation</term><term>Cellular stress</term><term>Cepaea nemoralis</term><term>Cerebral ischemia</term><term>Chick</term><term>Chick embryos</term><term>Collateral</term><term>Collateral blood</term><term>Committee guidelines</term><term>Conventional treatments</term><term>Cytoprotection</term><term>Daily exposures</term><term>Development challenge fund</term><term>Dicarlo</term><term>Drosophila melanogaster</term><term>Eld</term><term>Electromagnetic</term><term>Embryo</term><term>Energy stores</term><term>Free oxygen</term><term>Free oxygen radicals</term><term>Free radicals</term><term>Frequency electromagnetic</term><term>Frequency range</term><term>Gene activity</term><term>Gene expression</term><term>Gene expression levels</term><term>Goodman</term><term>Grant sponsor</term><term>Guideline</term><term>Gutzeit</term><term>Health care</term><term>Health research</term><term>Heat shock</term><term>Heat shock protein</term><term>Heat shock proteins</term><term>Heat shock response</term><term>Heat stress</term><term>Hela cells</term><term>High sars</term><term>Hsps</term><term>Human cells</term><term>Human leukocytes</term><term>Hypoxia</term><term>Increase none</term><term>Inducible form</term><term>Infarct size</term><term>Ischemia</term><term>Ischemiareperfusion</term><term>Ischemiareperfusion injuries</term><term>Ischemic</term><term>Ischemic conditions</term><term>Ischemic injuries</term><term>Ischemic injury</term><term>Ischemic insult</term><term>Ischemic penumbra</term><term>Ischemic strokes</term><term>Ischemic tissue</term><term>Katp channels</term><term>Land snail</term><term>Lawson health research institute</term><term>Leukocyte</term><term>Litovitz</term><term>Lower sars</term><term>Many studies</term><term>Maximal expression</term><term>Metabolic byproducts</term><term>Metal shielding</term><term>Microwave exposure</term><term>Mild stress</term><term>Mobile phone radiation</term><term>Mrna</term><term>Mussel immunocyte</term><term>Myocardial cells</term><term>Nascent proteins</term><term>Nematode</term><term>Nematode caenorhabditis elegans</term><term>Neurosci lett shupak</term><term>Nitric</term><term>Nitric oxide</term><term>Ontario research</term><term>Opioid</term><term>Opioid receptors</term><term>Opioids</term><term>Other proteins</term><term>Other systems</term><term>Oxide</term><term>Pain thresholds</term><term>Physiol</term><term>Prato</term><term>Preconditioning</term><term>Preconditioning studies</term><term>Promoter</term><term>Protective effect</term><term>Protective effects</term><term>Protective proteins</term><term>Protein synthesis</term><term>Receptor</term><term>Repair damage</term><term>Reperfusion</term><term>Reperfusion injury</term><term>Reperfusion insult</term><term>Reperfusion stage</term><term>Rest period</term><term>Right vessel</term><term>Robertson</term><term>Sars</term><term>Short term</term><term>Shupak</term><term>Small heat shock proteins</term><term>Small hsps</term><term>Stress proteins</term><term>Stress response</term><term>Stroke foundation</term><term>Superoxide</term><term>Superoxide radicals</term><term>Thrombolytic agents</term><term>Transgenic</term><term>Transgenic studies</term><term>Wound healing</term></keywords></textClass></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">Ischemia‐reperfusion injuries, such as those suffered from various types of cardiovascular disease, are major causes of death and disability. For relatively short periods of ischemia, much of the damage is potentially reversible and in fact, does not occur until the influx of oxygen during the reperfusion stage. Because of this, there is a window of opportunity to protect the ischemic tissue. Here, we review several mechanisms of protection, such as heat shock proteins, opioids, collateral blood flow, and nitric oxide induction, and the evidence indicating that magnetic fields may be used as a means of providing protection via each of these mechanisms. While there are few studies demonstrating direct protection with magnetic field therapies, there are a number of published reports indicating that electromagnetic fields may be able to influence some of the biochemical systems with protective applications. Bioelectromagnetics © 2006 Wiley‐Liss, Inc.</div></front></TEI><affiliations><list><country><li>Canada</li></country></list><tree><country name="Canada"><noRegion><name sortKey="Robertson, John A" sort="Robertson, John A" uniqKey="Robertson J" first="John A." last="Robertson">John A. Robertson</name></noRegion><name sortKey="Bureau, Yves" sort="Bureau, Yves" uniqKey="Bureau Y" first="Yves" last="Bureau">Yves Bureau</name><name sortKey="Prato, Frank S" sort="Prato, Frank S" uniqKey="Prato F" first="Frank S." last="Prato">Frank S. Prato</name><name sortKey="Prato, Frank S" sort="Prato, Frank S" uniqKey="Prato F" first="Frank S." last="Prato">Frank S. Prato</name><name sortKey="Robertson, John A" sort="Robertson, John A" uniqKey="Robertson J" first="John A." last="Robertson">John A. Robertson</name><name sortKey="Thomas, Alex W" sort="Thomas, Alex W" uniqKey="Thomas A" first="Alex W." last="Thomas">Alex W. Thomas</name><name sortKey="Thomas, Alex W" sort="Thomas, Alex W" uniqKey="Thomas A" first="Alex W." last="Thomas">Alex W. Thomas</name><name sortKey="Thomas, Alex W" sort="Thomas, Alex W" uniqKey="Thomas A" first="Alex W." last="Thomas">Alex W. Thomas</name><name sortKey="Thomas, Alex W" sort="Thomas, Alex W" uniqKey="Thomas A" first="Alex W." last="Thomas">Alex W. Thomas</name></country></tree></affiliations></record>Pour manipuler ce document sous Unix (Dilib)
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